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Brill, A. L., Fischer, T.T., Walters, J.M., Marlier, A., Sewanan, L.R., Robert, M.E., Wilson, P.C., Chung, H.J., Campbell, S.G., Cantley, L.G., and Ehrlich, B.E. (2020). "Polycystin 2 is increased in disease to protect against stress-induced cell death." Sci Rep. 10(1): 386.
Perry, R.J., Zhang, D., Guerra, M.T., Brill, A.L., Goedeke, L., Nasiri, A.R., Rabin-Court, A., Wang, Y., Peng, L., Dufour, S., Zhang, Y., Zhang, X.M., Butrico, G.M., Toussaint, K., Nozaki, Y., Cline, G.W., Petersen, K.F., Nathanson, M.H., Ehrlich, B.E., and Shulman, G.I. (2020). “Glucagon stimulates gluconeogenesis by InsP3R-I mediated hepatic lipolysis.” Nature 579(7798): 279-283.
Grosshans, H.K., Fischer, T.T., Steinle, J.A., Brill, A.L., and Ehrlich, B.E. (2020). “Neuronal Calcium Sensor 1 is up-regulated in response to stress to promote cell survival and motility in cancer cells.” Mol Oncol. doi: 10.1002/1878-0261.12678.
Ng, R., Sewanan, L.R., Brill, A.L., Stankey, P., Li, X., Qyang, Y., Ehrlich, B.E., and Campbell, S.G. (2020). "Contractile work directly modulates mitochondrial protein levels in human engineered heart tissues." American journal of physiology. Heart and circulatory physiology 318(6): H1516-h1524.
Brill, A.L.*, Kuo, I.Y*., Lemos, F.O., Jiang, J.Y., Falcone, J.L., Kimmerling, E.P., Cai, Y., Dong, K., Kaplan, D.L., Wallace, D.P., Hofer, A.M., and Ehrlich, B.E. (2019). “Polycystin 2 regulates mitochondrial calcium signaling, bioenergetics, and dynamics through mitofusin 2.” Sci Signaling. 12, eaat7397.
Neuman, J.C., Schaid, M.D., Brill, A.L., Fenske, R.J., Kibbe, C.R., Fontaine, D.A., Sdao, S.M., Brar, H.K., Connors, K.M., Wienkes, H.N., Eliceiri, K.W., Merrins, M.J., Davis, D.B., and Kimple, M.E. (2017). “Enriching Islet Phospholipids with Eicosapentaenoic Acid Reduces Prostaglandin E2 Signaling and Enhances Diabetic b-Cell Function.” Diabetes. 66(6):1572-1585.
Fenske, R.J., Cadena, M.T., Harenda, Q.E., Wienkes, H.N., Carbajal, K., Schaid, M.D., Laundre, E., Brill, A.L., Truchan, N.A., Brar, H.K., Wisinski, J.A., Jinjin, C., Graham, T.E., Engin, F., and Kimple, M.E. (2017). “The Inhibitory G-protein α-subunit, Gαz, promotes Type 1 diabetes-like pathophysiology in NOD mice.” Endocrinology. 158(6):1645-1658.
Brill, A.L., Wisinski, J.A., Cadena, M.T., Thompson, M.F., Fenske, R.J., Brar, H.K., Schaid, M.D., Pasker, R.L., and Kimple, M.E. (2016). “Synergy Between Gαz Deficiency and GLP-1 Analog Treatment in Preserving Functional b-Cell Mass in Experimental Diabetes.” Mol Endocrinol. 30(5):543-556.
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